Saturday, December 27, 2008

Disappointing decision on U.S. beef petition i.e. FORCE FEEDING KOREANS USDA MAD COW BEEF

[Editorial] Disappointing decision on U.S. beef petition

The Constitutional Court has ruled against a constitutional petition by 96,000 citizens claiming that the “process by which the Lee Myung-bak administration resumed imports of American beef by formalizing the move with an official announcement (gosi) in the daily government gazette (Gwanbo) was unconstitutional.” The Gwanbo is equivalent to the Federal Register in the United States. Its decision is a big disappointment for the Koreans who had hoped the court would stand in defense of the people’s safety and quarantine inspection sovereignty, because if the gosi was declared unconstitutional in accordance with the petition, which had more signatories than any in history, it would have been automatically invalidated and the Korean government would then have had the justification to renegotiate the beef deal with the United States.

The constitutional petition put before the Constitutional Court asserted that the administration’s move “significantly increases the possibility of bovine spongiform encephalopathy in humans and therefore infringes on constitutionally-guaranteed dignity and worth as humans, the right to pursue happiness, the right to life, and the right to health.” Negotiations with the United States on beef made it so that Korea accepts American beef without limits to age or parts, and that Korea cannot halt beef imports even if there is an outbreak of mad cow disease in the United States. Additional negotiations allowed for restrictions on beef from cows older than 30 months of age and on specified risk material, or SRM, but that was not a fundamental solution. It wrongly assumed that the United States had strengthened prohibitions on animal-based feed that had been relaxed, and another mistake was permitting the importation of some SRM, believing it to be parts that are safe.

Even the current head of the Ministry of Government Legislation said that the process was constitutionally flawed and that he, too, would have taken the issue to the Constitutional Court had he been in the opposition. He said that it is highly problematic, in a constitutional way, to implement something with a directive announcement (gosi) from the relevant Cabinet minister without legislation-drafting procedures, when the issue directly relates to the country’s health.

“While the gosi’s protection measures may not be perfect,” the court said it its decision, “one cannot conclude that this was wholly, or very much, an action that violated the state's constitutional duty to protect the safety of lives and bodies.” It is hard to understand how surrendering quarantine inspection sovereignty and exposing the country to the risk of mad cow disease cannot be concluded to be in inconsistent with or inadequate as far as the duty to the state. It is also disappointing that the court never even had an open oral debate that included expert testimony about a case that was the subject of intense national interest.

The mission of the Constitutional Court is to defend the basic rights of the people and weed out laws that are unconstitutional for violating those rights. The minority opinion was right in stating that the gosi “violates the petitioners’ basic rights because it inadequately carries out the state’s duty to protect basic rights regarding (the safety) of life and body.”

Please direct questions or comments to [englishhani@hani.co.kr]


http://english.hani.co.kr/arti/english_edition/e_editorial/329968.html




Tuesday, May 13, 2008 Concerned Americans against Mad Cow Disease STATEMENT OF SOLIDARITY with Koreans May 13, 2008

snip...

Re..Korea vs USDA beef (the truth)

Greetings KIWA et al and all Koreans,

A kind greetings from Bacliff, Texas.

I submit this data with great concern, sincerity, and in peace.

President Lee Myung-bak states ;

Greetings KIWA et al and all Koreans,

A kind greetings from Bacliff, Texas.

I submit this data with great concern, sincerity, and in peace.

President Lee Myung-bak states ;

"halt imports if another case of bovine spongiform encephalopathy turned up in the United States." now that is funny. a mad cow with BSE would have to drop from the sky onto the white house lawn and run around jerking, trembling, stumbling and staggering uncontrollably for months before anyone would every confirm a mad cow case with BSE, and then it would take an act of congress to confirm it, and President Lee Myung-bak knows this. it's all about commodities and futures $$$

Monday, May 5, 2008

STATEMENT OF DR. RICHARD RAYMOND USDA UNDERSECRETARY FOR FOOD SAFETY Regarding the Safety of the U.S. Food Supply please see full text with some additional information the good Dr. Raymond seems to have forgotten about ;



http://usdameatexport.blogspot.com/2008/05/statement-of-dr-richard-raymond-usda.html



USDA BEEF DEAL WITH KOREA AND the truth, what the usda et al are not telling you...



http://flounder068.vox.com/library/post/usda-beef-deal-with-korea-and-the-truth-what-the-usda-et-al-are-not-telling-you.html



DOWNER CATTLE SCHOOL LUNCH PROGRAM




http://downercattle.blogspot.com/



http://downercattle.blogspot.com/2008/05/humane-society-releases-new-video-of.html




"Atypical forms of BSE have emerged which, although rare, appear to be more virulent than the classical BSE that causes vCJD."

Sincerely, Pierluigi Gambetti, MD Director, National Prion Disease Pathology Surveillance Center Stephen M. Sergay, MB, BCh President, American Academy of Neurology



http://www.aan.com/news/?event=read&article_id=4397&page=72.45.45



THE LAST TWO MAD COWS documentED IN TEXAS AND ALABAMA WERE ATYPICAL BSE. then GW, USDA et al shut the testing down $$$ (new they would find more. ...tss)

please see full text with additional comments and links @ ;



http://prionunitusaupdate2008.blogspot.com/



Sunday, March 16, 2008

MAD COW DISEASE terminology UK c-BSE (typical), atypical BSE H or L, and or Italian L-BASE



http://bse-atypical.blogspot.com/2008/03/mad-cow-disease-terminology-uk-c-bse.html



==============================

THE only fool is one who fools himself, and GW and his administration and their junk science will fool humans for just so long, then the incubation will catch up. none of this was about science, it was all about commodities and futures and the exporting of beef. nothing else mattered, literally, just ask old stanley prusiner the nobel prize winner for the PRION, what did old stan say ;

STANLEY PRUSINER NOBEL PEACE PRIZE WINNER ON THE PRION

US AG SEC AND LAYCRAFT

"nothing matters, except beef from Canada under 30 months bone in beef product, that's ALL THAT MATTERS!" US SENATOR AND STAN THE MAN SLAM USDA "DAMNING TESTIMONY"

Senator Michael Machado from California

"USDA does not know what's going on". "USDA is protecting the industry". " SHOULD the state of California step in"

Stanley Prusiner

"nobody has ever ask us to comment"

"they don't want us to comment"

"they never ask"

i tried to see Venemon, after Canadian cow was discovered with BSE. went to see lyle. after talking with him.

absolute ignorance.

then thought i should see Venemon.

it was clear his entire policy was to get cattle boneless beef prods across the border.

nothing else mattered.

his aids confirmed this.

5 times i tried to see Venemon, never worked.

eventually met with carl rove the political.

he is the one that arranged meeting with Venemon.

just trying to give you a sense of the distance.

threat to health public safety.

was never contacted.

yes i believe that prions are bad to eat and you can die from them.END

Dr. Stan bashing Ann Veneman - 3 minutes - Damning testimony



http://maddeer.org/video/embedded/08snip.ram



File Name: USDA DON'T ASK DON'T TELL POLICY 02snip.rpm DAMNING testimony of consumer consumption of Washington mad cow in California



http://www.maddeer.org/video/embedded/02snip.rm



In this context, a word is in order about the US testing program. After the discovery of the first (imported) cow in 2003, the magnitude of testing was much increased, reaching a level of >400,000 tests in 2005 (Figure 4). Neither of the 2 more recently indigenously infected older animals with nonspecific clinical features would have been detected without such testing, and neither would have been identified as atypical without confirmatory Western blots. Despite these facts, surveillance has now been decimated to 40,000 annual tests (USDA news release no. 0255.06, July 20, 2006) and invites the accusation that the United States will never know the true status of its involvement with BSE.



http://www.cdc.gov/ncidod/EID/vol12no12/06-0965.htm



see full text ;



http://sciencebushwhacked.blogspot.com/



10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. MBM IN COMMERCE USA 2007

Date: March 21, 2007 at 2:27 pm PST REASON Blood meal used to make cattle feed was recalled because it was cross-contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement. VOLUME OF PRODUCT IN COMMERCE 42,090 lbs. DISTRIBUTION WI

REASON Products manufactured from bulk feed containing blood meal that was cross contaminated with prohibited meat and bone meal and the labeling did not bear cautionary BSE statement. VOLUME OF PRODUCT IN COMMERCE 9,997,976 lbs. DISTRIBUTION ID and NV

END OF ENFORCEMENT REPORT FOR MARCH 21, 2007



http://www.fda.gov/bbs/topics/enforce/2007/ENF00996.html



Subject: MAD COW FEED RECALL USA SEPT 6, 2006 1961.72 TONS IN COMMERCE AL, TN, AND WV Date: September 6, 2006 at 7:58 am PST

snip...

see listings and references of enormous amounts of banned mad cow protein 'in commerce' in 2006 and 2005 ; Friday, April 25, 2008

Substances Prohibited From Use in Animal Food or Feed [Docket No. 2002N-0273] (Formerly Docket No. 02N-0273) RIN 0910-AF46



http://madcowfeed.blogspot.com/2008/04/substances-prohibited-from-use-in.html



2006 was a banner year too for mad cow protein fed out into commerce. those were just one of many ; Specified Risk Materials



http://madcowspontaneousnot.blogspot.com/2008/02/specified-risk-materials-srm.html



NOR-98 ATYPICAL SCRAPIE USA UPDATE AS AT OCT 2007



http://nor-98.blogspot.com/




http://www.yacomitservice.com/php/eng/board.php?board=kkkqna&command=body&no=84



snip...



http://usdavskorea.blogspot.com/2008/05/concerned-americans-against-mad-cow.html



USDA VS KOREA typical or atypical BSe



http://usdavskorea.blogspot.com/



Friday, May 9, 2008 USDA VS KOREA typical or atypical BSe Honorable people of Korea,

a kind and warm greetings from Texas.

snip...



http://usdavskorea.blogspot.com/2008/05/usda-vs-korea-typical-or-atypical-bse.html



Why Americans, As Well as Koreans, Should Be Worried About Mad Cow Tainted USA Beef

By Terry S. Singeltary Sr. May 15, 2008

Straight to the Source

Web Note: This is an important commentary by Terry S. Singeltary Sr., on a recent Business Week story on the controversy in South Korea over their government's lifting on the ban on conventional (non-organic) beef, despite the fact that the USDA is still allowing slaughterhouse waste and blood and manure to be fed to cows, and refusing to test all cows at slaughter. See the Mad Cow section of the OCA website for in-depth information. Terry is a regular blogger on the OCA website on Mad Cow issues.

Ronnie Cummins

One Korean official says the probability of a human being catching a mad cow disease by eating U.S. beef is like the one of a golf player scoring a hole-in-one and then being killed by lightning.

this is typical BSe. you here industry groups comment 'your more likely to get hit by a car than die from CJD'. well, maybe so, but my mother and many more did not die from getting hit by a car, they died from CJD, my mothers being the hvCJD (confirmed), and my neighbors mother died from CJD (confirmed). the UKBSEnvCJD _only_ theory is incorrect. there are more strains of mad cow than the UK BSE in beef to nvCJD in humans in the UK. The deception by the USDA, FDA, and the Bush administration about mad cow disease, CJD, and all Transmissible Spongiform Encephalopathy over the past 8 years have been outrageous, to a point of being criminal. I am vested in nothing, but the truth.

snip...see full text ;



http://www.grassrootsnetroots.org/articles/article_12387.cfm



Tuesday, May 13, 2008

Concerned Americans against Mad Cow Disease STATEMENT OF SOLIDARITY with Koreans May 13, 2008



http://usdavskorea.blogspot.com/2008/05/concerned-americans-against-mad-cow.html



http://flounder068.vox.com/library/post/concerned-americans-against-mad-cow-disease-statement-of-solidarity-with-koreans-may-13-2008.html


http://www.koreantopnews.com/story.php?title=USDA_VS_KOREA_typical_or_atypical_BSe_Concerned_Americans_against_Mad_Cow_Disease_STATEMENT_OF_SOLIDARITY_with_Koreans_May_13_2008



BSE YOUNGEST AGE STATISTICS UNDER 30 MONTHS



http://bseyoungestage.blogspot.com/



http://flounder068.vox.com/library/post/bse-youngest-age-statistics-under-30-months.html



Friday, June 20, 2008

USDA TO KOREA AND THE WORLD, EAT THAT AND LIKE IT



http://usdavskorea.blogspot.com/2008/06/usda-to-korea-and-world-eat-that-and.html



Wednesday, June 11, 2008

OIE Recognition of the BSE Status of Members RESOLUTION No. XXI (Adopted by the International Committee of the OIE on 27 May 2008)



snip...



http://usdavskorea.blogspot.com/2008/06/oie-recognition-of-bse-status-of.html



http://organicconsumers.org/forum/index.php?showtopic=1566



U.S. slams door on revising S. Korea beef import pact

June 11, 2008, 10:14PM



http://usdavskorea.blogspot.com/2008/06/us-slams-door-on-revising-s-korea-beef.html



Saturday, June 7, 2008

Export Requirements for the Republic of Korea IMPORT HEALTH REQUIREMENTS FOR U.S. BEEF AND BEEF PRODUCTS



http://usdavskorea.blogspot.com/2008/06/export-requirements-for-republic-of.html



Wednesday, July 23, 2008

Audit says USDA lost track of imported cattle Report No. 50601-0012-Ch March 2008 Audit says USDA lost track of imported cattle Canada has reported 13 cases of mad cow



http://usdameatexport.blogspot.com/




UPDATE DECEMBER 2008


Monday, December 22, 2008

[Docket No. FDA–2008–D–0597] Draft Guidance for Industry: Small Entities Compliance Guide for Renderers—Substances Prohibited From Use in Animal Food

Greetings,

I kindly wish to submit the following to [Docket No. FDA–2008–D–0597] ;

I would kindly like to once again comment on the failed attempts of the FDA et al to stop the spread of animal TSEs, including BSE, through the legal, and illegal feeding practices, of feeding animal protein to livestock for human and animal consumption. Since the terribly flawed, partial, and voluntary August 4, 1997 ruminant to ruminant feed ban was put into place, literally 100s of thousands of tons of banned animal protein has been fed out into commerce, even as late as 2007, when some 10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. Blood meal used to make cattle feed was recalled because it was cross-contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement. NOW, how much of that product that went out into commerce was fed out to cattle, and how much was ever recovered ? AND to think that feeding blood to livestock producing animals is still legal, when scientific study after study shows that TSEs are easily transmitted via blood. IT is absolutely unacceptable that still in 2008, the USA is still feeding highly suspect mad cow feed to USA cattle, and other livestock producing animals. Especially when the last two cases of BSE that were allowed to be tested and reported were of the atypical BSE category, of which we now know the atypical BSE is more virulent than that of the typical BSE, and when ARS research on the atypical BSE said long ago the SRM rules may need to be changed, IF the atypical BSE were to be proven to be more virulent. Why do we continue to flounder? I have submitted to these BSE feed dockets until I am blue in the face, and still to date, they still debate an issue that should have been settled long ago. IT's a fine example of how big ag, big industry, have a stranglehold on sound science and policy making thereof. How many millions of animals and humans have been needlessly exposed to this TSE agent, due to nothing more than ignorance and greed, simply because of a disease that is 100% fatal, but one that has such a long incubation period. For the government and industry as a whole, to continue to flagrantly violate said rules and regulations, in my opinion, should be regarded as criminal, and treated as such. People are dying. ...

Please see references ;

10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. MBM IN COMMERCE USA 2007

Date: March 21, 2007 at 2:27 pm PST

REASON

Blood meal used to make cattle feed was recalled because it was cross-contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement. VOLUME OF PRODUCT IN COMMERCE 42,090 lbs. DISTRIBUTION WI

REASON

Products manufactured from bulk feed containing blood meal that was cross contaminated with prohibited meat and bone meal and the labeling did not bear cautionary BSE statement. VOLUME OF PRODUCT IN COMMERCE 9,997,976 lbs. DISTRIBUTION ID and NV

END OF ENFORCEMENT REPORT FOR MARCH 21, 2007



http://www.fda.gov/bbs/topics/enforce/2007/ENF00996.html





NEW URL



http://www.fda.gov/Safety/Recalls/EnforcementReports/2007/ucm120446.htm



2006 WAS A BANNER YEAR ALSO FOR MAD COW FEED BAN VIOLATIONS ''IN COMMERCE''

Subject: MAD COW FEED RECALL USA SEPT 6, 2006 1961.72 TONS IN COMMERCE AL, TN, AND WV Date: September 6, 2006 at 7:58 am PST

snip...

see listings and references of enormous amounts of banned mad cow protein 'in commerce' in 2006 and 2005 ;

see full text ;

Friday, April 25, 2008

Substances Prohibited From Use in Animal Food or Feed [Docket No. 2002N-0273] (Formerly Docket No. 02N-0273) RIN 0910-AF46



http://madcowfeed.blogspot.com/2008/04/substances-prohibited-from-use-in.html



SPECIFIED RISK MATERIALS



http://madcowspontaneousnot.blogspot.com/2008/02/specified-risk-materials-srm.html



0C3.01

Transmission of atypical BSE to Microcebus murinus, a non-human primate: Development of clinical symptoms and tissue distribution of PrPres

Background: Atypical BSE cases have been observed in Europe, Japan and North America. They differ in their PrPres profiles from those found in classical BSE. These atypical cases fall into 2 types, depending on the molecular mass of the unglycosylated PrPres band observed by Western blot: the L -type (lower molecular mass than the typical BSE cases) and H-type (higher molecular mass than the typical BSE cases).

Objectives and Methods: In order to see if the atypical BSE cases were transmissible to primates, either animals (were intracerebrally inoculated with 50 ul of a 10% brain homogenates of two atypical French BSE case, a H-type (2 males and 2 females) and a L-type (2 males and 2 females).

Results: Only one of the four lemurs challenged with H-type BSE died without clinical signs after 19 months post inoculation (mpi), whereas all the 4 animals inoculated with L -type BSE died at 19 mpi (2 males) and 22 mpi (2 females). Three months before their sacrifice, they developed blindness, tremor, abnormal posture, incoordinated movements, balance loss. Symptoms got worse according to the disease progression, until severe ataxia. The brain tissue were biochemically and immunocytochemically investigated for PrPres. For the H-type, spongiform changes without PrPres accumulation were observed in the brainstem. However Western blot analysis did not allow to detect PrPres into the brain. For the L-type, severe spongiosis was evidenced into the thalamus, the striatum, the mesencephalon, and the brainstem. whereas into the cortex the spongiosis was evidenced, but the Vacuolisation was weaker. Strong deposits of PrPres were detected by western blot, PET-blot and immunocytochemistry in the CNS: dense accumulation was observed into the thalamus, the striatum, and the hippocampus whereas in the cerebral cortex, PrPres was prominently accumulated in plaques. Western blot analysis also readily confirmed the presence of protease-resistant prion protein.

Conclusions: L-type infected lemurs showed survival times considerably shorter than for classical BSE strain, indicating that the disease is caused by a very virulent distinct prion strain in a model of non human primate.



http://www.neuroprion.org/resources/pdf_docs/conferences/prion2008/abstract-book-prion2008.pdf



look at the table and you'll see that as little as 1 mg (or 0.001 gm) caused 7% (1 of 14) of the cows to come down with BSE;

Risk of oral infection with bovine spongiform encephalopathy agent in primates

Corinne Ida Lasmézas, Emmanuel Comoy, Stephen Hawkins, Christian Herzog, Franck Mouthon, Timm Konold, Frédéric Auvré, Evelyne Correia, Nathalie Lescoutra-Etchegaray, Nicole Salès, Gerald Wells, Paul Brown, Jean-Philippe Deslys

Summary The uncertain extent of human exposure to bovine spongiform encephalopathy (BSE)--which can lead to variant Creutzfeldt-Jakob disease (vCJD)--is compounded by incomplete knowledge about the efficiency of oral infection and the magnitude of any bovine-to-human biological barrier to transmission. We therefore investigated oral transmission of BSE to non-human primates. We gave two macaques a 5 g oral dose of brain homogenate from a BSE-infected cow. One macaque developed vCJD-like neurological disease 60 months after exposure, whereas the other remained free of disease at 76 months. On the basis of these findings and data from other studies, we made a preliminary estimate of the food exposure risk for man, which provides additional assurance that existing public health measures can prevent transmission of BSE to man.

snip...

BSE bovine brain inoculum 100 g 10 g 5 g 1 g 100 mg 10 mg 1 mg 0·1 mg 0·01 mg Primate (oral route)* 1/2 (50%) Cattle (oral route)* 10/10 (100%) 7/9 (78%) 7/10 (70%) 3/15 (20%) 1/15 (7%) 1/15 (7%) RIII mice (ic ip route)* 17/18 (94%) 15/17 (88%) 1/14 (7%) PrPres biochemical detection The comparison is made on the basis of calibration of the bovine inoculum used in our study with primates against a bovine brain inoculum with a similar PrPres concentration that was inoculated into mice and cattle.8 *Data are number of animals positive/number of animals surviving at the time of clinical onset of disease in the first positive animal (%). The accuracy of bioassays is generally judged to be about plus or minus 1 log. ic ip=intracerebral and intraperitoneal. Table 1: Comparison of transmission rates in primates and cattle infected orally with similar BSE brain inocula

Published online January 27, 2005



http://www.thelancet.com/journal/journal.isa



P04.27

Experimental BSE Infection of Non-human Primates: Efficacy of the Oral Route

Holznagel, E1; Yutzy, B1; Deslys, J-P2; Lasmézas, C2; Pocchiari, M3; Ingrosso, L3; Bierke, P4; Schulz-Schaeffer, W5; Motzkus, D6; Hunsmann, G6; Löwer, J1 1Paul-Ehrlich-Institut, Germany; 2Commissariat à l´Energie Atomique, France; 3Instituto Superiore di Sanità, Italy; 4Swedish Institute for Infectious Disease control, Sweden; 5Georg August University, Germany; 6German Primate Center, Germany

Background:

In 2001, a study was initiated in primates to assess the risk for humans to contract BSE through contaminated food. For this purpose, BSE brain was titrated in cynomolgus monkeys.

Aims:

The primary objective is the determination of the minimal infectious dose (MID50) for oral exposure to BSE in a simian model, and, by in doing this, to assess the risk for humans. Secondly, we aimed at examining the course of the disease to identify possible biomarkers.

Methods:

Groups with six monkeys each were orally dosed with lowering amounts of BSE brain: 16g, 5g, 0.5g, 0.05g, and 0.005g. In a second titration study, animals were intracerebrally (i.c.) dosed (50, 5, 0.5, 0.05, and 0.005 mg).

Results:

In an ongoing study, a considerable number of high-dosed macaques already developed simian vCJD upon oral or intracerebral exposure or are at the onset of the clinical phase. However, there are differences in the clinical course between orally and intracerebrally infected animals that may influence the detection of biomarkers.

Conclusions:

Simian vCJD can be easily triggered in cynomolgus monkeys on the oral route using less than 5 g BSE brain homogenate. The difference in the incubation period between 5 g oral and 5 mg i.c. is only 1 year (5 years versus 4 years). However, there are rapid progressors among orally dosed monkeys that develop simian v CJD as fast as intracerebrally inoculated animals.

The work referenced was performed in partial fulfillment of the study “BSE in primates“ supported by the EU (QLK1-2002-01096).



http://www.prion2007.com/pdf/Prion%20Book%20of%20Abstracts.pdf



Calves were challenged by mouth with homogenised brain from confirmed cases of BSE. Some received 300g (3 doses of 100g), some 100g, 10g or 1g. They were then left to develop BSE, but were not subjected to the normal stresses that they might have encountered in a dairy herd. Animals in all four groups developed BSE. There has been a considerable spread of incubation period in some of the groups, but it appears as if those in the 1 and 10g challenge groups most closely fit the picture of incubation periods seen in the epidemic. Experiments in progress indicate that oral infection can occur in some animals with doses as low as 0.01g and 0.001g. .........



http://www.defra.gov.uk/animalh/bse/science-research/pathog.html#dose



It is clear that the designing scientists must also have shared Mr Bradley's surprise at the results because all the dose levels right down to 1 gram triggered infection.



http://www.bseinquiry.gov.uk/files/ws/s145d.pdf



6. It also appears to me that Mr Bradley's answer (that it would take less than say 100 grams) was probably given with the benefit of hindsight; particularly if one considers that later in the same answer Mr Bradley expresses his surprise that it could take as little of 1 gram of brain to cause BSE by the oral route within the same species. This information did not become available until the "attack rate" experiment had been completed in 1995/96. This was a titration experiment designed to ascertain the infective dose. A range of dosages was used to ensure that the actual result was within both a lower and an upper limit within the study and the designing scientists would not have expected all the dose levels to trigger infection. The dose ranges chosen by the most informed scientists at that time ranged from 1 gram to three times one hundred grams. It is clear that the designing scientists must have also shared Mr Bradley's surprise at the results because all the dose levels right down to 1 gram triggered infection.



http://www.bseinquiry.gov.uk/files/ws/s147f.pdf



snip... full text ;



http://madcowfeed.blogspot.com/2008/12/docket-no-fda2008d0597-draft-guidance.html



Evaluation of the Human Transmission Risk of an Atypical Bovine Spongiform Encephalopathy Prion Strain

Qingzhong Kong,1* Mengjie Zheng,1 Cristina Casalone,2 Liuting Qing,1 Shenghai Huang,1? Bikram Chakraborty,1 Ping Wang,1 Fusong Chen,1 Ignazio Cali,1 Cristiano Corona,2 Francesca Martucci,2 Barbara Iulini,2 Pierluigi Acutis,2 Lan Wang,1 Jingjing Liang,1 Meiling Wang,1 Xinyi Li,1 Salvatore Monaco,3 Gianluigi Zanusso,3 Wen-Quan Zou,1 Maria Caramelli,2 and Pierluigi Gambetti1* Department of Pathology, Case Western Reserve University, Cleveland, Ohio 44106,1 CEA, Istituto Zooprofilattico Sperimentale, 10154 Torino, Italy,2 Department of Neurological and Visual Sciences, University of Verona, 37134 Verona, Italy3 *Corresponding author. Mailing address: Department of Pathology, Case Western Reserve University, Cleveland, OH 44106. Phone for Pierluigi Gambetti: (216) 368-0586. Fax: (216) 368-2546. E-mail: mhtml:%7B33B38F65-8D2E-434D-8F9B-8BDCD77D3066%7Dmid://00000025/!x-usc:mailto:pxg13@case.edu . Phone for Qingzhong Kong: (216) 368-1756. Fax: (216) 368-2546. E-mail: mhtml:%7B33B38F65-8D2E-434D-8F9B-8BDCD77D3066%7Dmid://00000025/!x-usc:mailto:qxk2@case.edu ?Present address: Department of Patient Education and Health Information, Cleveland Clinic Foundation, Cleveland, OH 44195. Received November 30, 2007; Accepted January 16, 2008.

Bovine spongiform encephalopathy (BSE), the prion disease in cattle, was widely believed to be caused by only one strain, BSE-C. BSE-C causes the fatal prion disease named new variant Creutzfeldt-Jacob disease in humans. Two atypical BSE strains, bovine amyloidotic spongiform encephalopathy (BASE, also named BSE-L) and BSE-H, have been discovered in several countries since 2004; their transmissibility and phenotypes in humans are unknown. We investigated the infectivity and human phenotype of BASE strains by inoculating transgenic (Tg) mice expressing the human prion protein with brain homogenates from two BASE strain-infected cattle. Sixty percent of the inoculated Tg mice became infected after 20 to 22 months of incubation, a transmission rate higher than those reported for BSE-C. A quarter of BASE strain-infected Tg mice, but none of the Tg mice infected with prions causing a sporadic human prion disease, showed the presence of pathogenic prion protein isoforms in the spleen, indicating that the BASE prion is intrinsically lymphotropic. The pathological prion protein isoforms in BASE strain-infected humanized Tg mouse brains are different from those from the original cattle BASE or sporadic human prion disease. Minimal brain spongiosis and long incubation times are observed for the BASE strain-infected Tg mice. These results suggest that in humans, the BASE strain is a more virulent BSE strain and likely lymphotropic.



http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=2268471





Thursday, December 04, 2008 2:37 PM

"we have found that H-BSE can infect humans."

personal communication with Professor Kong. ...TSS



"the biochemical signature of PrPres in the BASE-inoculated animal was found to have a higher proteinase K sensitivity of the octa-repeat region. We found the same biochemical signature in three of four human patients with sporadic CJD and an MM type 2 PrP genotype who lived in the same country as the infected bovine." ... interesting. ... TSS

Thursday, June 05, 2008

Review on the epidemiology and dynamics of BSE epidemics

Vet. Res. (2008) 39:15 www.vetres.org DOI: 10.1051/vetres:2007053 c INRA, EDP Sciences, 2008 Review article

snip...

And last but not least, similarities of PrPres between Htype BSE and human prion diseases like CJD or GSS have been put forward [10], as well as between L-type BSE and CJD [17]. These findings raise questions about the origin and inter species transmission of these prion diseases that were discovered through the BSE active surveillance.

snip...

Cases of atypical BSE have only been found in countries having implemented large active surveillance programs. As of 1st September 2007, 36 cases (16 H, 20 L) have been described all over the world in cattle: Belgium (1 L) [23], Canada (1 H)15, Denmark (1 L)16, France (8 H, 6 L)17, Germany (1 H, 1 L) [13], Italy (3 L)18, Japan (1 L) [71], Netherlands (1 H, 2 L)19, Poland (1 H, 6 L)20, Sweden (1 H)21, United Kingdom (1 H)22, and USA (2 H)23. Another H-type case has been found in a 19 year old miniature zebu in a zoological park in Switzerland [56]. It is noteworthy that atypical cases have been found in countries that did not experience classical BSE so far, like Sweden, or in which only few cases of classical BSE have been found, like Canada or the USA.

And last but not least, similarities of PrPres between Htype BSE and human prion diseases like CJD or GSS have been put forward [10], as well as between L-type BSE and CJD [17]. These findings raise questions about the origin and inter species transmission of these prion diseases that were discovered through the BSE active surveillance.

full text 18 pages ;



http://www.vetres.org/index.php?option=article&access=standard&Itemid=129&url=/articles/vetres/pdf/2008/04/v07232.pdf



please see full text ;



http://bse-atypical.blogspot.com/2008/06/review-on-epidemiology-and-dynamics-of.html



***Atypical forms of BSE have emerged which, although rare, appear to be more virulent than the classical BSE that causes vCJD.***

Progress Report from the National Prion Disease Pathology Surveillance Center

An Update from Stephen M. Sergay, MB, BCh & Pierluigi Gambetti, MD

April 3, 2008



http://www.aan.com/news/?event=read&article_id=4397&page=72.45.45



In this context, a word is in order about the US testing program. After the discovery of the first (imported) cow in 2003, the magnitude of testing was much increased, reaching a level of >400,000 tests in 2005 (Figure 4). Neither of the 2 more recently indigenously infected older animals with nonspecific clinical features would have been detected without such testing, and neither would have been identified as atypical without confirmatory Western blots. Despite these facts, surveillance has now been decimated to 40,000 annual tests (USDA news release no. 0255.06, July 20, 2006) and invites the accusation that the United States will never know the true status of its involvement with BSE.

In short, a great deal of further work will need to be done before the phenotypic features and prevalence of atypical BSE are understood. More than a single strain may have been present from the beginning of the epidemic, but this possibility has been overlooked by virtue of the absence of widespread Western blot confirmatory testing of positive screening test results; or these new phenotypes may be found, at least in part, to result from infections at an older age by a typical BSE agent, rather than neonatal infections with new "strains" of BSE. Neither alternative has yet been investigated.



http://www.cdc.gov/ncidod/EID/vol12no12/06-0965.htm



Wednesday, August 20, 2008

Bovine Spongiform Encephalopathy Mad Cow Disease typical and atypical strains, was there a cover-up ?



http://bse-atypical.blogspot.com/2008/08/bovine-spongiform-encephalopathy-mad.html



A New Prionopathy OR more of the same old BSe and sporadic CJD



http://creutzfeldt-jakob-disease.blogspot.com/2008/08/new-prionopathy-or-more-of-same-old-bse.html



Communicated by: Terry S. Singeltary Sr.

[In submitting these data, Terry S. Singeltary Sr. draws attention to the steady increase in the "type unknown" category, which, according to their definition, comprises cases in which vCJD could be excluded. The total of 26 cases for the current year (2007) is disturbing, possibly symptomatic of the circulation of novel agents. Characterization of these agents should be given a high priority. - Mod.CP]



http://pro-med.blogspot.com/2007/11/proahedr-prion-disease-update-2007-07.html



http://www.promedmail.org/pls/askus/f?p=2400:1001:6833194127530602005::NO::F2400_P1001_BACK_PAGE,F2400_P1001_PUB_MAIL_ID:1010,39963



There is a growing number of human CJD cases, and they were presented last week in San Francisco by Luigi Gambatti(?) from his CJD surveillance collection.

He estimates that it may be up to 14 or 15 persons which display selectively SPRPSC and practically no detected RPRPSC proteins.



http://www.fda.gov/ohrms/dockets/ac/06/transcripts/1006-4240t1.htm



http://www.fda.gov/ohrms/dockets/ac/06/transcripts/2006-4240t1.pdf



sporadic Fatal Familial Insomnia



http://sporadicffi.blogspot.com/



JOURNAL OF NEUROLOGY

MARCH 26, 2003

RE-Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob

disease in the United States

Email Terry S. Singeltary:



mailto:flounder9@verizon.net


I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to comment on the CDC's attempts to monitor the occurrence of emerging forms of CJD. Asante, Collinge et al [1] have reported that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest sporadic CJD. However, CJD and all human TSEs are not reportable nationally. CJD and all human TSEs must be made reportable in every state and internationally. I hope that the CDC does not continue to expect us to still believe that the 85%+ of all CJD cases which are sporadic are all spontaneous, without route/source. We have many TSEs in the USA in both animal and man. CWD in deer/elk is spreading rapidly and CWD does transmit to mink, ferret, cattle, and squirrel monkey by intracerebral inoculation. With the known incubation periods in other TSEs, oral transmission studies of CWD may take much longer. Every victim/family of CJD/TSEs should be asked about route and source of this agent. To prolong this will only spread the agent and needlessly expose others. In light of the findings of Asante and Collinge et al, there should be drastic measures to safeguard the medical and surgical arena from sporadic CJDs and all human TSEs. I only ponder how many sporadic CJDs in the USA are type 2 PrPSc?



http://www.neurology.org/cgi/eletters/60/2/176#535



THE PATHOLOGICAL PROTEIN

Hardcover, 304 pages plus photos and illustrations. ISBN 0-387-95508-9

June 2003

BY Philip Yam

CHAPTER 14 LAYING ODDS

Answering critics like Terry Singeltary, who feels that the U.S. under- counts CJD, Schonberger conceded that the current surveillance system has errors but stated that most of the errors will be confined to the older population.



http://www.thepathologicalprotein.com/



Diagnosis and Reporting of Creutzfeldt-Jakob Disease Singeltary, Sr et al. JAMA.2001; 285: 733-734. Vol. 285 No. 6, February 14, 2001 JAMA

Diagnosis and Reporting of Creutzfeldt-Jakob Disease

To the Editor: In their Research Letter, Dr Gibbons and colleagues1 reported that the annual US death rate due to Creutzfeldt-Jakob disease (CJD) has been stable since 1985. These estimates, however, are based only on reported cases, and do not include misdiagnosed or preclinical cases. It seems to me that misdiagnosis alone would drastically change these figures. An unknown number of persons with a diagnosis of Alzheimer disease in fact may have CJD, although only a small number of these patients receive the postmortem examination necessary to make this diagnosis. Furthermore, only a few states have made CJD reportable. Human and animal transmissible spongiform encephalopathies should be reportable nationwide and internationally.

Terry S. Singeltary, Sr Bacliff, Tex

1. Gibbons RV, Holman RC, Belay ED, Schonberger LB. Creutzfeldt-Jakob disease in the United States: 1979-1998. JAMA. 2000;284:2322-2323. FREE FULL TEXT



http://jama.ama-assn.org/cgi/content/extract/285/6/733?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&fulltext=singeltary&searchid=1&FIRSTINDEX=0&resourcetype=HWCIT



http://jama.ama-assn.org/cgi/content/full/285/6/733?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&fulltext=singeltary&searchid=1&FIRSTINDEX=0&resourcetype=HWCIT



2 January 2000 British Medical Journal U.S. Scientist should be concerned with a CJD epidemic in the U.S., as well



http://www.bmj.com/cgi/eletters/320/7226/8/b#6117



15 November 1999 British Medical Journal vCJD in the USA * BSE in U.S.



http://www.bmj.com/cgi/eletters/319/7220/1312/b#5406



Creutzfeldt Jakob Disease



http://creutzfeldt-jakob-disease.blogspot.com/



USA PRION UNIT BLOG



http://prionunitusaupdate2008.blogspot.com/



Sunday, April 20, 2008 Progress Report from the National Prion Disease Pathology Surveillance Center April 3, 2008

Atypical forms of BSE have emerged which, although rare, appear to be more virulent than the classical BSE that causes vCJD.

see full text ;



http://prionunitusaupdate2008.blogspot.com/2008/04/progress-report-from-national-prion.html



CJD TEXAS (cjd clusters)



http://cjdtexas.blogspot.com/



USA WRITTEN CJD QUESTIONNAIRE ???



http://cjdquestionnaire.blogspot.com/



Attending Dr.: Date / Time Admitted : 12/14/97 1228

UTMB University of Texas Medical Branch Galveston, Texas 77555-0543 (409) 772-1238 Fax (409) 772-5683 Pathology Report

FINAL AUTOPSY DIAGNOSIS Autopsy' Office (409)772-2858

FINAL AUTOPSY DIAGNOSIS

I. Brain: Creutzfeldt-Jakob disease, Heidenhain variant.



http://creutzfeldt-jakob-disease.blogspot.com/2008/07/heidenhain-variant-creutzfeldt-jakob.html



2007

The statistical incidence of CJD cases in the United States has been revised to reflect that there is one case per 9000 in adults age 55 and older. Eighty-five percent of the cases are sporadic, meaning there is no known cause at present.



http://www.cjdfoundation.org/fact.html




Friday, August 29, 2008

CREEKSTONE VS USDA COURT OF APPEALS, BUSH SAYS, NO WAY, NO HOW



http://madcowtesting.blogspot.com/2008/08/creekstone-vs-usda-court-of-appeals.html




Sunday, March 16, 2008

MAD COW DISEASE terminology UK c-BSE (typical), atypical BSE H or L, and or Italian L-BASE



http://bse-atypical.blogspot.com/2008/03/mad-cow-disease-terminology-uk-c-bse.html




HUMAN and ANIMAL TSE Classifications i.e. mad cow disease and the UKBSEnvCJD only theory JUNE 2008

snip...

Tissue infectivity and strain typing of the many variants Manuscript of the human and animal TSEs are paramount in all variants of all TSE. There must be a proper classification that will differentiate between all these human TSE in order to do this. With the CDI and other more sensitive testing coming about, I only hope that my proposal will some day be taken seriously. ...

snip...



http://cjdmadcowbaseoct2007.blogspot.com/2008/06/human-and-animal-tse-classifications-ie.html




0C3.01

Transmission of atypical BSE to Microcebus murinus, a non-human primate: Development of clinical symptoms and tissue distribution of PrPres

Background: Atypical BSE cases have been observed in Europe, Japan and North America. They differ in their PrPres profiles from those found in classical BSE. These atypical cases fall into 2 types, depending on the molecular mass of the unglycosylated PrPres band observed by Western blot: the L -type (lower molecular mass than the typical BSE cases) and H-type (higher molecular mass than the typical BSE cases).

Objectives and Methods: In order to see if the atypical BSE cases were transmissible to primates, either animals (were intracerebrally inoculated with 50 ul of a 10% brain homogenates of two atypical French BSE case, a H-type (2 males and 2 females) and a L-type (2 males and 2 females).

Results: Only one of the four lemurs challenged with H-type BSE died without clinical signs after 19 months post inoculation (mpi), whereas all the 4 animals inoculated with L -type BSE died at 19 mpi (2 males) and 22 mpi (2 females). Three months before their sacrifice, they developed blindness, tremor, abnormal posture, incoordinated movements, balance loss. Symptoms got worse according to the disease progression, until severe ataxia. The brain tissue were biochemically and immunocytochemically investigated for PrPres. For the H-type, spongiform changes without PrPres accumulation were observed in the brainstem. However Western blot analysis did not allow to detect PrPres into the brain. For the L-type, severe spongiosis was evidenced into the thalamus, the striatum, the mesencephalon, and the brainstem. whereas into the cortex the spongiosis was evidenced, but the Vacuolisation was weaker. Strong deposits of PrPres were detected by western blot, PET-blot and immunocytochemistry in the CNS: dense accumulation was observed into the thalamus, the striatum, and the hippocampus whereas in the cerebral cortex, PrPres was prominently accumulated in plaques. Western blot analysis also readily confirmed the presence of protease-resistant prion protein.

Conclusions: L-type infected lemurs showed survival times considerably shorter than for classical BSE strain, indicating that the disease is caused by a very virulent distinct prion strain in a model of non human primate.



http://www.neuroprion.org/resources/pdf_docs/conferences/prion2008/abstract-book-prion2008.pdf



P7.09

Biochemical screening for identification of atypical bse in belgium, 1999-present

Authors

Alexandre DobIy: Caroline Rodeghiero, Riet Geeroms; Stephanie Durand, Jessica De Sloovere, Emanuel Yanopdenbosch, Stefan Roels,

Content

Background: Recently atypical forms of BSE have been described. Western blot analyses showed that, in comparison to the classic BSE (C-type), they are demonstrable by a higher or lower molecular weight of the unglycosylated PrPres. They Viere thus named H-type and L-type BSE (L-type is also called BASE). In addition they show a lower proportion of diglycosylated PrPres than C-type. These emerging types represent different strains of BSE. They show unique incubation periods and histological lesions. Such types have been described on different continents. Indeed they might correspond to "sporadic" forms of BSE. In 2004 we already described one L-type in Belgium.

Objective: We retrospectively analysed the bovines at least 7-year-old in the Belgian archive of BSE ­diagnosed cattle in order to determine the prevalence of the two types of atypical BSE in Belgium.

Methods: We analysed homogenates from 39 bovines of 93 months old in median (min: 84, max: 181 months). The most recent one was diagnosed in 2006. We used Western blot with a panel of anti-PrP antibodies (Ab). They detect different regions of the PrP protein, from N-terminal to C-terminal: 12B2, 9A2, Sha31. SAFB4, 94B4. Their combination is aimed at an efficient typing diagnostic. We detected bound Ab with SuperSignal West Dura (Pierce) and analysed PrPres, signals with an image-analysis software (Quantity One, Bio-Rad).

Results: The results are still under analysis. We will detail the most crucial characteristics for typing PrPres. These include 1) the apparent molecular mass of the an-, mono- and diglycosylated bands, 2) the binding affinity to the five Ab (e.g.12B2 for H-type), 3) the presence of a fourth (unglycosylated) band and 4) the glycoprofile based on the relative proportions of the visible bands.

Discussion: The emergence of atypical types of BSE is partially due to a better knowledge of prion strains and more efficient diagnostic techniques. As the area in the brain where the PrPres is deposited can differ drastically between the types, it is essential to ascertain that the sampling techniques and analyses are adapted to these new types. As these new strains seem more virulent than classic types, they represent one of the next challenges in the field of prions.



http://www.neuroprion.org/resources/pdf_docs/conferences/prion2008/abstract-book-prion2008.pdf



http://www.prion2008.com/



Tuesday, November 11, 2008

Transmission of atypical bovine prions to mice transgenic for human prion protein

DOI: 10.3201/eid1412.080941



http://bse-atypical.blogspot.com/2008/11/transmission-of-atypical-bovine-prions.html



Wednesday, August 20, 2008

Bovine Spongiform Encephalopathy Mad Cow Disease typical and atypical strains, was there a cover-up ?



http://bse-atypical.blogspot.com/2008/08/bovine-spongiform-encephalopathy-mad.html



Tuesday, June 3, 2008

SCRAPIE USA UPDATE JUNE 2008 NOR-98 REPORTED PA



http://nor-98.blogspot.com/2008/06/scrapie-usa-update-june-2008-nor-98.html



SCRAPIE USA



http://scrapie-usa.blogspot.com/



Sunday, September 07, 2008

CWD LIVE TEST, and the political aspects or fallout of live testing for BSE in cattle in the USA



http://chronic-wasting-disease.blogspot.com/2008/09/cwd-live-test-and-political-aspects-or.html



Saturday, October 18, 2008

WYOMING STAR VALLEY MOOSE TESTS POSITIVE FOR CWD



http://chronic-wasting-disease.blogspot.com/2008/10/wyoming-star-valley-moose-tests.html



http://chronic-wasting-disease.blogspot.com/



Friday, December 12, 2008

The prion strain phenomenon: Molecular basis and unprecedented features



http://bse-atypical.blogspot.com/2008/12/prion-strain-phenomenon-molecular-basis.html



Docket Management Docket: 02N-0276 - Bioterrorism Preparedness; Registration of Food Facilities, Section 305 Comment Number: EC -254 Accepted - Volume 11



http://www.fda.gov/OHRMS/DOCKETS/DOCKETS/02n0276/02N-0276-EC-254.htm



http://www.fda.gov/ohrms/dockets/dockets/02n0276/02N-0276-EC-254.htm



Docket Management Docket: 02N-0273 - Substances Prohibited From Use in

Animal Food or Feed; Animal Proteins Prohibited in Ruminant Feed

Comment Number: EC -10

Accepted - Volume 2



http://www.fda.gov/ohrms/dockets/dailys/03/Jan03/012403/8004be07.html



PART 2



http://www.fda.gov/ohrms/dockets/dailys/03/Jan03/012403/8004be09.html



Monday, December 08, 2008

vCJD & dental treatment



http://creutzfeldt-jakob-disease.blogspot.com/2008/12/vcjd-dental-treatment.html



Friday, December 12, 2008

Prions in Milk from Ewes Incubating Natural Scrapie



http://scrapie-usa.blogspot.com/2008/12/prions-in-milk-from-ewes-incubating.html



Friday, December 05, 2008

Detection of Prion Infectivity in Fat Tissues of Scrapie-Infected Mice



http://scrapie-usa.blogspot.com/2008/12/detection-of-prion-infectivity-in-fat.html



REPORT ON CURRENT & FUTURE SURVEILLANCE FOR BOVINE SPONGIFORM ENCEPHALOPATHY



http://madcowtesting.blogspot.com/2008/11/report-on-current-future-surveillance.html



November 25, 2008

Update On Feed Enforcement Activities To Limit The Spread Of BSE



http://madcowfeed.blogspot.com/2008/11/november-2008-update-on-feed.html



Plasma & Serum Proteins Receive Continued FDA Approval

4/25/2008 APC, Inc. is pleased to advise our customers and industry partners that as anticipated, the Food and Drug Administration (FDA) will continue to allow the use of bovine blood, plasma and serum proteins in ruminant feeds.

In April 2008 FDA announced the publication of its Final Rule for 21 CFR Part 589.2001 - Substances Prohibited From Use in Animal Food or Feed. FDA specifically stated in their opinion that, "FDA is not prohibiting the use of blood and blood products in animal feed because we believe such a prohibition would do very little to reduce the risk of BSE transmission."

Known as a leader in developing nutritional products for the swine industry, where 95% of pig starter diets in the United States contain functional proteins, APC has more recently developed their line of colostrum replacers, supplements, feed additives and milk replacer ingredients for calves. Products include plasma, serum and immunoglobulin concentrate based Acquire®, Lifeline®, Gammulin® and Nutrapro® used to optimize the health and performance of calves.

To view the full report for Final Rule 21 CFR Part 589.2001 visit:



http://www.fda.gov/OHRMS/DOCKETS/98fr/FDA-2002-N-0031-nfr.pdf



To view the complete Feed Rule 21 CFR Part 589 visit:



http://www.accessdata.fda.gov/scripts/cdrh/cfdocs/cfcfr/CFRSearch.cfm?CFRPart=589&showFR=1



Friday, November 21, 2008

Plasma & Serum Proteins Receive Continued FDA Approval



http://madcowfeed.blogspot.com/2008/11/plasma-serum-proteins-receive-continued.html



http://madcowfeed.blogspot.com/



Thursday, November 27, 2008

Prion diseases are efficiently transmitted by blood transfusion in sheep



http://vcjdblood.blogspot.com/2008/11/prion-diseases-are-efficiently.html



stupid is, as stupid does. (forest gump)

Thank You,

I am sincerely,

Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA 77518

Friday, August 8, 2008

South Koreans Fill Streets of Seoul to Continue Protest Against US Beef Imports

Sent: Thursday, August 07, 2008 2:14 PM Subject: South Koreans Fill Streets of Seoul to Continue Protest Against US Beef Imports

South Koreans Fill Streets of Seoul to Continue Protest Against US Beef Imports

For the past two months, protesters have been filling the streets of Seoul condemning a decision to lift a ban on imported beef from the United States. We speak with Michael Hansen, senior scientist for Consumers Union. He is in Seoul, where he is testifying before the South Korean National Assembly at a special committee hearing on mad cow disease.

For the past two months protesters have been filling the streets of Seoul condemning a decision to lift a ban on imported beef from the United States. On Wednesday I talked Michael Hansen in Seoul, senior scientist for Consumers Union. He is in Seoul where he is testifying before the South Korean National Assembly at a special committee hearing on mad cow disease.

Michael Hansen, senior scientist for Consumers Union. He speaks to us from Seoul, where he is testifying before the South Korean National Assembly at a special committee hearing on BSE.

LISTEN WATCH

Real Video Stream

http://i1.democracynow.org/2008/8/7/south_koreans_fill_streets_of_seoul


Real Audio Stream

http://play.rbn.com/?url=demnow/demnow/demand/2008/aug/audio/dn20080807.ra&proto=rtsp&start=53:11


MP3 Download

More…

http://i1.democracynow.org/2008/8/7/south_koreans_fill_streets_of_seoul


Saturday, June 7, 2008

Export Requirements for the Republic of Korea IMPORT HEALTH REQUIREMENTS FOR U.S. BEEF AND BEEF PRODUCTS

http://usdavskorea.blogspot.com/2008/06/export-requirements-for-republic-of.html


Why Americans, As Well as Koreans, Should Be Worried About Mad Cow Tainted USA Beef

By Terry S. Singeltary Sr. May 15, 2008

Straight to the Source

Web Note: This is an important commentary by Terry S. Singeltary Sr., on a recent Business Week story on the controversy in South Korea over their government's lifting on the ban on conventional (non-organic) beef, despite the fact that the USDA is still allowing slaughterhouse waste and blood and manure to be fed to cows, and refusing to test all cows at slaughter. See the Mad Cow section of the OCA website for in-depth information. Terry is a regular blogger on the OCA website on Mad Cow issues.

Ronnie Cummins

One Korean official says the probability of a human being catching a mad cow disease by eating U.S. beef is like the one of a golf player scoring a hole-in-one and then being killed by lightning.

this is typical BSe. you here industry groups comment 'your more likely to get hit by a car than die from CJD'. well, maybe so, but my mother and many more did not die from getting hit by a car, they died from CJD, my mothers being the hvCJD (confirmed), and my neighbors mother died from CJD (confirmed). the UKBSEnvCJD _only_ theory is incorrect. there are more strains of mad cow than the UK BSE in beef to nvCJD in humans in the UK. The deception by the USDA, FDA, and the Bush administration about mad cow disease, CJD, and all Transmissible Spongiform Encephalopathy over the past 8 years have been outrageous, to a point of being criminal. I am vested in nothing, but the truth.

snip...see full text ;

http://www.grassrootsnetroots.org/articles/article_12387.cfm


http://yacomitservice.com/php/eng/board.php?board=kkkqna&page=4&command=body&no=84


http://www.designtv.co.kr/tc/101


Tuesday, May 13, 2008

Concerned Americans against Mad Cow Disease STATEMENT OF SOLIDARITY with Koreans May 13, 2008

http://usdavskorea.blogspot.com/2008/05/concerned-americans-against-mad-cow.html


http://flounder068.vox.com/library/post/concerned-americans-against-mad-cow-disease-statement-of-solidarity-with-koreans-may-13-2008.html


http://www.pressian.com/Scripts/section/article.asp?article_num=60080519121918


http://blog.etnews.co.kr/nanofood/137892


http://news.msn.co.kr/article/read.html?cate_code=1200&article_id=200805191405451200


http://yallang.blogspot.com/2008/05/what-would-i-like-to-read.html


http://www.koreantopnews.com/story.php?title=USDA_VS_KOREA_typical_or_atypical_BSe_Concerned_Americans_against_Mad_Cow_Disease_STATEMENT_OF_SOLIDARITY_with_Koreans_May_13_2008


BSE YOUNGEST AGE STATISTICS UNDER 30 MONTHS

http://bseyoungestage.blogspot.com/


http://flounder068.vox.com/library/post/bse-youngest-age-statistics-under-30-months.html


[Source: New York Times] ????

[Source: Organic Consumers Association] ????

Why Koreans Still Reject U.S. Beef: Stop the Madness By Michael Hansen The New York Times, June 20, 2008

OpEd by Michael Hansen, a senior scientist at Consumers Union

THE Korean beef market, once the third-largest import!er of American beef, has shut its doors to the United States. Why? Because Koreans are worried about eating meat tainted with mad cow disease, which can be fatal to humans. Recent attempts by Korea's president, Lee Myung-bak, to reopen the market have brought tens of thousands of demonstrators to the streets in protest.

American beef producers could easily allay those fears by subjecting every cow at slaughter to the so-called rapid test, which costs about $20 per carcass and screens for this brain-wasting disease in a few hours rather than days. But the United States Department of Agriculture won't allow that.

In 2004, Creekstone Farms in Arkansas City, Kan., wanted to test the cattle it slaughters to comply with the wishes of its Korean and Japanese customers. But the department ruled that the rapid test could only be used as part of its own mad cow surveillance program, which randomly checks about 1 in 1,000 dead and slaughtered cattle in the United States every year. The sale of the kits to private companies is prohibited under an obscure 1913 law that allows the department to prohibit veterinary products that it considers "worthless."

Creekstone sued the government in 2006, arguing in court that the Agriculture Department could not deem worthless a test that it used in its own surveillance program. The court agreed, but the department appealed. A decision is expected soon.

It is hard to understand why the Agriculture Department wants to stand in the way. Yes, the test has limitations: it can miss a case of mad cow disease, also called bovine spongiform encephalopathy, in the very early stages of incubation. But it can catch the disease in later stages, before animals show symptoms. Between 2001 and 2006, the European Union used the test to turn up 1,117 cases of mad cow disease in seemingly healthy cattle approved for slaughter.

Ideally, the Agriculture Department would follow the rules set up in Europe and Japan that require every cow over a certain age to be tested before being slaughtered. At the very least the department should not prevent private companies from testing.

Companies that use the rapid test should also be allowed to label their meat as having been "tested for mad cow" for American consumers who would like this extra level of protection. A Consumers Union national survey done in January 2004 found that 71 percent of adults who eat beef would pay more to support testing, and of those, 95 percent were willing to spend 10 cents more per pound for tested meat.

In the Creekstone case, the Agriculture Department argued that the tests should be prohibited because if one company started using them, consumer demand would drive all companies to use them, and that would add to the price of beef. But would that be such a bad thing? Isn't this how the laws of supply and demand are supposed to work?

Most Americans, like Koreans, understand that testing for mad cow could save lives - and they'd like to have that option.

Comments flounder Today, 10:57 AM

From: "Terry S. Singeltary Sr." To: Sent: Tuesday, June 17, 2008 3:20 PM Subject: [BSE-L] Beef Import!s to Korea: An Open Letter to President Bush Korean middle school student Chae-song Kim asks that the trade agreement be reconsidered

Beef Import!s to Korea: An Open Letter to President Bush Korean middle school student Chae-song Kim asks that the trade agreement be reconsidered

Chae-song Kim (internews)

Published 2008-06-14 17:04 (KST)

Dear Mr. President,

Hello. I am an ordinary Korean teenager but I am not sending you this letter for an ordinary reason. I wanted to talk about something very serious with you. It is about the new agreement between the Republic of Korea and the United States.

Our countries are now both very sensitive about the beef issue. My belief is that free trade must be fair trade, as US ambassadors are on record as saying in the FTA between Peru, which means that both countries should benefit.

I admit that Korea has benefitted from trade in the areas of automobile exports, IT products, and cell phones. However, Koreans are very upset with their government. This is clear from the candlelight demonstrations in Korea, yet our government is saying that it is not possible to renegotiate the KORUS FTA agreement because of the federal government.

This is a photo that I took from the Seoul Plaza Hotel. There were more than 700,000 people gathered there. The news reported that only 19.7 percent said "yes" to President Lee. The rule about the trade agreement says, roughly, that within 20 days of the signing, if many citizens say "no" to the agreement, it is possible to make changes. What would be the problem to change the agreement now?

If the US and the Korean government do not accept people's opinions, the next generation of Koreans will have a negative image of the United States. We are trying not to buy products made in the United States and we do not respect our President. In the long term, demonstrations by Koreans will cause more damage to the US than changing this agreement would.

I've read that on March 20, 1996, BSE (Bovine Spongiform Encephalopathy) spread in the UK because prions are not destroyed until heated to 600 degrees Celsius.

This tells us that there is a high possibility of BSE infection in humans because even if humans cook their beef products thoroughly, prions are not going to be destroyed. Even the US Congress is saying that Canadian beef is dangerous and that the US shouldn't import! Canadian beef aged over 30 months.

Even if Congress still says that Koreans have no proof about the dangers of BSE in US beef, we should err on the side of caution and emphasize prevention. Why would the European Union say "no", completely, to US beef? Why would the US itself not consume beef older than 30 months? The truth is that there is a possibility of it being very dangerous.

BSE has spread since the 1980s. Scientists say that it is caused by the use of animal feed made from cows that already have the disease. Since then, for 20 years, in countries all around the world, cows in countries like the United Kingdom, have consumed such feed and BSE became a serious problem. Along with Europe, in 2001 Japan detected BSE in cattle and in 2003, even the US discovered BSE in its stocks.

Regardless of this, I've been told that the United States is still allowing the production of feed based on cow parts. Also, without any outstanding symptoms, the cow is butchered and allowed to be exported. With this in mind, would Korean citizens feel safe about import!ed US beef?

I want to ask you: Wouldn't this agreement be beneficial to the US only in the short term? Couldn't this agreement be perceived as violating the rights of Koreans to feel safe? Please consider the result that this agreement could and will leave, both now and for the future.

I am considering going to an Ivy League college. If I succeed, and even if I don't, children in the States are my friends. Their family is my family. I just want my family in Korea and my US family to be healthy. I hope you’ll be remembered as a president who cared for citizens' health.

A concerned Korean student,

Chae-song Kim

http://english.ohmynews.com/articleview/article_view.asp?at_code=434590


Hello Chae-song Kim and the Honorable concerned Korean student,

Believe in what your heart is telling you. Your Countryman and Countrywoman should be proud of your stance to protect your people. Please continue your fight for the truth, as the truth will set us all free. ...TSS

full text ;

http://english.ohmynews.com/TALK_BACK/bbs_view.asp?mscssid=&ba_code=63&ba_status=&cur_page=&bb_page=1&bb_ord=N&bb_code=537170&bbsh_gb=S&bbsh_string=


June 11, 2008, 10:14PM

U.S. slams door on revising S. Korea beef

import! pact Pressed at home, Seoul had wanted 30-month limit on age of the cattle

http://www.chron.com/disp/story.mpl/nation/5832282.html


http://usdavskorea.blogspot.com/2008/06/us-slams-door-on-revising-s-korea-beef.html


well, so the U.S. slams the mad cow door on Korea and it's people, due to the bungled beef deal the USDA shoved down Lee's throat. sadly, Lee signed the deal oblivious to what really has been going on behind closed doors for years here in the USA, and the USDA et al knew they had a fish on the line. my God, this guy was totally ignorant of what they were doing. now the kind honorable people of Korea will be force fed USDA certified beef. beef that has been highly suspect of mad cow disease since the USDA shut down testing, this after finding two cases of the atypical BSE in Alabama and Texas. remember, atypical BSE is more virulent than the UK BSE strain.

Also, it seems the O.I.E. has sealed the deal on trading all strains of TSE i.e. mad cow disease strains globally, all for a buck, commodities and futures, to hell with human health. it's business as usual folks, eat up, and die old and demented, if your lucky. I must apologize to the kind honorable citizens of Korea for what my Government has done. I tried.

But as the USDA certified beef starts to flood Korean markets, remember one thing Korea, you don't have to buy it. let it rot, until the USDA et al gets there head out of their pockets, and start to test all food producing cattle and all livestock for BSE and all TSE.

CJD is a slow death while incubating. so you will not see the body bags all at once. as in the past here, it will be labeled as dementia, misdiagnosed as Alzheimer's and other dementia ailments. it will become an acceptable death as here in the USA due to the push by the industries and your Government due to the financial aspect of it.

The OIE and my Government sold their souls to the devil, and if you don't believe me, just read the history. let it all be sporadic and or spontaneous they say, and make them eat it, and like it. that's their motto. to hell with the consumer. Every American and Korean consumer should be demanding 100% BSE/TSE mad cow testing on all livestock food producing animals, for humans and animals. This should be a no-brainer, but instead, it's a brain eater.

Consumption of beef is but only one route of many, that can kill you from mad cow disease (all strains). friendly fire i.e. iatrogenic CJD from the medical and surgical arenas, dental, and blood, all are a real threat, from 2nd, 3rd, 4th passage, from someone that consumed meat from an animal with TSE.

They can be long incubators, not clinical yet, but they can go on and infected many more via these routes, so please do not get hung up on the 'hamburger only' route. this is one of many routes, from one of many strains, from only one species.

WE must take all Transmissible Spongiform Encephalopathies as the real threat they really are. DO NOT let the incubation period of all these documented TSEs in cattle, sheep, goats, deer, elk, mink, cats, do not let them fool you. The long term threat is very real, it's been real, and it's been ignored, all the while CJD is rising in the USA. all the time the USA has been shipping cattle and feed to God knows whom. again, it was the OIE, the USDA et al, and their BSE MRR (Minimal Risk Region) rule, that opened the gates to the exporting of all strains of TSE globally. This new rule set back the eradication BSE to the stone age, or back to day one in or around 1985, when BSE was first _documented_. The BSE MRR policy erased all attempted eradication of BSE. This will be one more of GWs et al sad, sad legacies that will go down in history as nothing more than what the UK did back when they failed to warn the world of their tainted cattle, MBM (greaves) etc. they just continued to ship it around the globe. BUT what the OIE, GW, and the USDA did was simply made it legal i.e. BSE MRR, the legal trading of all strains of TSE globally $$$

Wednesday, June 11, 2008

OIE Recognition of the BSE Status of Members RESOLUTION No. XXI (Adopted by the International Committee of the OIE on 27 May 2008)

snip...SEE FULL TEXT ;

http://blog.daum.net/kenrocks/5396197


Friday, June 20, 2008

USDA TO KOREA AND THE WORLD, EAT THAT AND LIKE IT

http://usdavskorea.blogspot.com/2008/06/usda-to-korea-and-world-eat-that-and.html


Friday, August 8, 2008

Texas Firm Recalls Cattle Heads That Contain Prohibited Materials SRMs 941,271 pounds with tonsils not completely removed

http://madcowfeed.blogspot.com/2008/08/texas-firm-recalls-cattle-heads-that.html


The statistical incidence of CJD cases in the United States has been _revised_ to reflect that there is

one case per 9000 in adults age 55 and older.

Eighty-five percent of the cases are sporadic, meaning there is no known cause at present.

http://www.cjdfoundation.org/fact.html


Wednesday, July 9, 2008

10 people killed by new CJD-like disease USA

http://cjdmadcowbaseoct2007.blogspot.com/2008/06/novel-human-disease-with-abnormal-prion.html


TSS

Wednesday, July 9, 2008

EXPORT OF BEEF AND BEEF PRODUCTS TO KOREA USDA FSIS NOTICE EXPIRES: 7/1/09 OPI: OPPD

UNITED STATES DEPARTMENT OF AGRICULTURE FOOD SAFETY AND INSPECTION SERVICE WASHINGTON, DC

FSIS NOTICE

45-08 7/9/08

DISTRIBUTION: Electronic

NOTICE EXPIRES: 7/1/09 OPI: OPPD

EXPORT OF BEEF AND BEEF PRODUCTS TO KOREA

I. PURPOSE This notice provides information about the export of beef and beef products to Korea. In particular, it sets out: - what products are eligible for export to Korea; - what products are ineligible for export to Korea; and - the certification requirements and that a Statement of Verification (SOV) letter is no longer required to accompany exported beef and beef products to Korea if the slaughter dates are on or after the establishment’s Quality System Assessment (QSA) Program approval date.

II. ELIGIBLE ESTABLISHMENTS AND PRODUCTS TO EXPORT TO KOREA A. Korean beef importers and U.S. exporters have reached a commercial understanding that, as a transitional measure, only U.S. beef from cattle less than 30- months of age will be shipped to Korea. The Agricultural Marketing Service (AMS) has set up a voluntary QSA Program to verify that beef from participating plants will be from cattle less than 30 months of age. Exporting establishments may choose to participate in the AMS QSA Program that verifies that the beef being certified is from cattle less than 30 months of age. 1. For establishments that participate in the AMS QSA Program, the following statement can appear in Remarks on the FSIS Form 9060-5: “The beef or beef products were produced at a verified establishment under the USDA Less than 30 Month Age Verification Quality System Assessment (QSA) Program for Korea.” 2. Participation in this program is not required for issuance of the FSIS 9060-5. However, at this time, Korea will not accept at port-of-entry shipments of beef without the QSA Program statement in the Remarks section of the FSIS 9060-5, and Korean 2

quarantine officials will return shipments without the statement to the owner/agent of the product. 3. A list of AMS QSA Program approved establishments and their approval dates can be obtained from the AMS website at: http://www.ams.usda.gov/AMSv1.0/ams.fetchTemplateData.do?template=TemplateM&n avID=GradingCertificationandVerfication&leftNav=GradingCertificationandVerfication&p age=QSAPProgram B. Eligible beef and beef products, including bone-in beef, deboned beef, offal, and variety meats, are to be derived from cattle slaughtered on or after the slaughter establishment's QSA Program approval date and need to be produced under an approved AMS Export Verification (EV) program for beef to Korea. Federally inspected establishments producing beef and beef products intended for export to Korea need to participate in an AMS, EV program and be listed on the "Official Listing of Eligible Suppliers for USDA Bovine EV Program". Inspection program personnel can access the list at: http://www.ams.usda.gov/AMSv1.0/getfile?dDocName=STELPRD3105269 NOTE: FSIS personnel can view eligible product lists by establishment in Outlook at: Public Folders/All Public Folders/OFO/Export Verification Program and on the FSIS intranet: https://inside.fsis.usda.gov/fsis/emp/static/partnerweb/index.jsp. C. In addition, deboned beef products from cattle less than 30 months of age that were processed at eligible establishments before October 5, 2007 are eligible. The beef is to have been produced according to the AMS EV program in place at the time of production. A list of these eligible establishments and their specific periods of eligibility are available at: http://www.ams.usda.gov/AMSv1.0/getfile?dDocName=STELPRD3105269. NOTE: Information about approval of warehouses, cold storages, or similar facilities that export beef and beef products to Korea can be obtained from FSIS Notice 39-07 (Jun 26, 2007; PDF only).

III. INELIGIBLE PRODUCTS Under the current EV program described in II.B. above, the following products are ineligible for export to Korea: 1. beef and beef products derived from cattle imported from Canada for immediate slaughter; 2. beef and beef products derived from cattle imported from Canada that were resident in the U.S. less than 100 days prior to slaughter; 3. beef and beef products derived from beef and beef products imported into the U.S. from third countries; and 4. Processed beef products. FSIS NOTICE 45-08 3

IV. LABELING REQUIREMENTS Labels applied to Porterhouse steaks and T-bone steaks are to include a notation that these cuts come from animals less than 30 months of age.

V. CERTIFICATION REQUIREMENTS A. Beef and beef products from eligible establishments with slaughter dates on or after the establishment’s QSA Program approval date are to be accompanied by: 1. FSIS 9060-5 (MPG series dated 07/19/2001 or MPH series dated 12/14/2006) Meat and Poultry Export Certificate of Wholesomeness. For establishments that participate in the AMS QSA Program, the following statement can appear in Remarks on the FSIS Form 9060-5: “The beef or beef products were produced at a verified establishment under the USDA Less than 30 Month Age Verification USDA Quality System Assessment (QSA) Program for Korea.” 2. FSIS Form 9305-7, dated 07/08/2008, Certificate for Export of Beef and Beef Products to the Republic of Korea. NOTE: Inspection program personnel can obtain this form in Outlook at: Public Folders/All Public Folders/Agency Issuances/Forms/FSIS 9000 Series. 3. For this product AMS will no longer provide a SOV letter, and such a letter is not required to issue export certification. B. Boneless beef from eligible establishments and with processing dates before October 5, 2007 are to be accompanied by: 1. FSIS 9060-5 (MPG series dated 07/19/2001 or MPH series dated 12/14/2006) Meat and Poultry Export Certificate of Wholesomeness. The following statement must be typed in the Remarks section: “These products were derived from cattle under 30 months of age at the time of slaughter and were slaughtered before October 5, 2007, in accordance with the EV program for Korea and stored in the United States.” 2. FSIS Form 9305-4 (06/07/2006), Certificate for Export of Beef and Beef Products to the Republic of Korea. NOTE: Inspection program personnel can obtain this form in Outlook at: Public Folders/All Public Folders/Agency Issuances/Forms/FSIS 9000 Series. 3. FSIS personnel can view eligible product lists by establishment in Outlook at: Public Folders/All Public Folders/OFO/Export Verification Program. 4. A Statement of Verification letter from AMS is required as part of the export certification process for product produced before October 5, 2007 (see FSIS Notice 4

http://www.fsis.usda.gov/OPPDE/rdad/FSISNotices/45-08.pdf

BSE YOUNGEST AGE STATISTICS UNDER 30 MONTHS

http://bseyoungestage.blogspot.com/

http://flounder068.vox.com/library/post/bse-youngest-age-statistics-under-30-months.html

----- Original Message ----- From: "Terry S. Singeltary Sr." <flounder9@VERIZON.NET> To: <BSE-L@LISTS.AEGEE.ORG> Sent: Wednesday, June 11, 2008 9:29 AM Subject: [BSE-L] OIE Recognition of the BSE Status of Members RESOLUTION No. XXI (Adopted by the International Committee of the OIE on 27 May 2008)

Wednesday, June 11, 2008

OIE Recognition of the BSE Status of Members RESOLUTION No. XXI (Adopted by the International Committee of the OIE on 27 May 2008)

snip...

CONSIDERING THAT

1. Adoption of subsequent Resolutions* since the 67th General Session of the OIE International Committee has established a procedure for annually updating a list of Members, categorised by their BSE risk according to the provisions of theTerrestrial Code,

2. During the 70th General Session, the International Committee adopted Resolution No. XVIII asking Members applying for a BSE risk evaluation to meet part of the costs sustained by the OIE Central Bureau in the evaluation process,

3. During the 72nd General Session, the OIE adopted Resolution No. XXI requesting the Director General to inform Delegates of Members whose country or zones are recognised with regard to their BSE risk status should annually confirm during the month of November whether their risk status and the criteria by which their status was recognised have remained unchanged,

4. Information published by the OIE is derived from declarations made by the official Veterinary Services of Members. The OIE is not responsible for inaccurate publication of a Member disease status based on inaccurate information, changes in epidemiological status or other significant events that were not promptly reported to the Central Bureau, subsequent to the time of declaration of the BSE risk status.

THE COMMITTEE

RESOLVES THAT

1. The Director General publish the following list of Members recognised as having a negligible BSE risk in accordance with Chapter 2.3.13. of the Terrestrial Code:

Australia, Argentina, Finland, Iceland, New Zealand, Norway, Paraguay, Singapore, Sweden and Uruguay.

2. The Director General publish the following list of Members recognised as having a controlled BSE risk in accordance with Chapter 2.3.13. of the Terrestrial Code:

Austria Belgium Brazil Canada Chile Chinese Taipei Cyprus Czech Republic Denmark Estonia France Germany Greece Hungary Ireland Italy Latvia Lichtenstein Lithuania Luxembourg Malta Mexico Netherlands Poland Portugal Slovak Republic Slovenia Spain Switzerland United Kingdom United States of America

AND

3. The Delegates of these Members will immediately notify the Central Bureau if BSE occurs in their countries or their territories.

_________

(Adopted by the International Committee of the OIE on 27 May 2008)

* 67th General Session (GS) Resolution No (Res) XVI and Res XI; 69th GS Res XV, and 71st GS Res XXII, 72nd GS Res XXIV and Res XXI..

http://www.oie.int/eng/info/en_statesb.htm?e1d6

IN A NUT SHELL ;

(Adopted by the International Committee of the OIE on 23 May 2006)

11. Information published by the OIE is derived from appropriate declarations made by the official Veterinary Services of Member Countries. The OIE is not responsible for inaccurate publication of country disease status based on inaccurate information or changes in epidemiological status or other significant events that were not promptly reported to the Central Bureau,

http://www.oie.int/eng/Session2007/RF2006.pdf

bought and paid for by your local cattle dealer $$$

IN my opinion the WOAH/OIE is nothing more than a organized bunch of lobbyist for the members Countries in support of there INDUSTRY, bound together as one, with the only purpose of open trade for there precious commodities and futures. Speaking only of BSE, they failed at every corner, and then just said to hell with it, well just trade all strains of TSE globally.

snip...

NOW, ask yourself why not one single mad cow has been documented in the USA since the Honorable Phyllis Fong of the OIG did the end around Johanns, Dehaven et al ??? found two atypical BSE or BASE cases and they flat shut it down i tell you. IF the OIE gives a favorable rating, IF the OIE gives any other rating but the lowest, poorest possible BSE/TSE rating, the OIE will have sealed there fate once and for all, because most of the world knows the truth about the USA and there mad cows. THE OIE will then be able to stand side by side with the USA, and proudly claim to have sold there soul to the devil, all for a buck, commodities and futures, to hell with human health. A 'CONTROLLED' RATING IS EXACTLY what the OIE will get if that is what they classify the USA as a 'CONTROLLED RATING'. IT will be controlled by Johanns, Dehaven, and GW. IT WILL BE RIGGED in other words. but that is nothing new, it's been rigged for years. ...

snip...SEE FULL TEXT with facts and sources @ ;

Wednesday, June 11, 2008

OIE Recognition of the BSE Status of Members RESOLUTION No. XXI (Adopted by the International Committee of the OIE on 27 May 2008)

http://usdavskorea.blogspot.com/2008/06/oie-recognition-of-bse-status-of.html

http://organicconsumers.org/forum/index.php?showtopic=1566

Saturday, June 7, 2008

Export Requirements for the Republic of Korea IMPORT HEALTH REQUIREMENTS FOR U.S. BEEF AND BEEF PRODUCTS

http://usdavskorea.blogspot.com/2008/06/export-requirements-for-republic-of.html

Why Americans, As Well as Koreans, Should Be Worried About Mad Cow Tainted USA Beef

By Terry S. Singeltary Sr. May 15, 2008

Straight to the Source

Web Note: This is an important commentary by Terry S. Singeltary Sr., on a recent Business Week story on the controversy in South Korea over their government's lifting on the ban on conventional (non-organic) beef, despite the fact that the USDA is still allowing slaughterhouse waste and blood and manure to be fed to cows, and refusing to test all cows at slaughter. See the Mad Cow section of the OCA website for in-depth information. Terry is a regular blogger on the OCA website on Mad Cow issues.

Ronnie Cummins

One Korean official says the probability of a human being catching a mad cow disease by eating U.S. beef is like the one of a golf player scoring a hole-in-one and then being killed by lightning.

this is typical BSe. you here industry groups comment 'your more likely to get hit by a car than die from CJD'. well, maybe so, but my mother and many more did not die from getting hit by a car, they died from CJD, my mothers being the hvCJD (confirmed), and my neighbors mother died from CJD (confirmed). the UKBSEnvCJD _only_ theory is incorrect. there are more strains of mad cow than the UK BSE in beef to nvCJD in humans in the UK. The deception by the USDA, FDA, and the Bush administration about mad cow disease, CJD, and all Transmissible Spongiform Encephalopathy over the past 8 years have been outrageous, to a point of being criminal. I am vested in nothing, but the truth.

snip...see full text ;

http://www.grassrootsnetroots.org/articles/article_12387.cfm

Tuesday, May 13, 2008

Concerned Americans against Mad Cow Disease STATEMENT OF SOLIDARITY with Koreans May 13, 2008

http://usdavskorea.blogspot.com/2008/05/concerned-americans-against-mad-cow.html

http://flounder068.vox.com/library/post/concerned-americans-against-mad-cow-disease-statement-of-solidarity-with-koreans-may-13-2008.html

http://www.koreantopnews.com/story.php?title=USDA_VS_KOREA_typical_or_atypical_BSe_Concerned_Americans_against_Mad_Cow_Disease_STATEMENT_OF_SOLIDARITY_with_Koreans_May_13_2008

Sunday, June 29, 2008 South Korea bans rallies against US beef imports

http://usdavskorea.blogspot.com/2008/06/south-korea-bans-rallies-against-us.html

South Korea ... whom PD Diary portrayed as having probably died of variant Creutzfeldt-Jakob disease (vCJD), the human form of mad cow disease, there is a moment when ... See all stories on this topic

ISN'T THIS WHAT THE U.S.A. DOCTOR AND MEDIA PUBLISHED FIRST ???

Portsmouth woman may have human form of Mad Cow Disease

06:38 PM EDT on Monday, April 7, 2008

Reported by: Wayne Carter PORTSMOUTH, Va. --

A 22-year-old Portsmouth woman is close to dying, and family says doctors believe the human equivalent of Mad Cow Disease could be the reason.

Creutzfeldt-Jakob Disease , or CJD, is so rare that there has only been one other possible case ever in the United States.

The Portsmouth Health Department is looking into the case because the variant form of the disease comes from eating infected meat, and Aretha Vincent's family says she's never left the United States.

http://www.wvec.com/news/portsmouth/stories/wvec_local_040708_mad_cow_disease.3fd7e5c7.html

please see full text ;

http://cjdmadcowbaseoct2007.blogspot.com/2008/06/portsmouth-woman-did-not-die-of-mad-cow.html

Wednesday, July 9, 2008 [Docket No. FDA-2008-N-0369] Ruminant Feed Ban Support Project; ``Response to RFA-FDA-08-008''

http://madcowfeed.blogspot.com/2008/07/docket-no-fda-2008-n-0369-ruminant-feed.html

MAD COW DISEASE terminology UK c-BSE (typical), atypical BSE H or L, and or Italian L-BASE

http://bse-atypical.blogspot.com/2008/03/mad-cow-disease-terminology-uk-c-bse.html

Please remember, the last two mad cows documented in the USA i.e. Alabama and Texas, both were of the 'atypical' BSE strain, and immediately after that, the USDA shut down the testing from 470,000 to 40,000 in the U.S. in 2007 out of about 35 million cattle slaughtered. also, science is showing that some of these atypical cases are more virulent to humans than the typical UK BSE strain ;

***Atypical forms of BSE have emerged which, although rare, appear to be more virulent than the classical BSE that causes vCJD.***

Progress Report from the National Prion Disease Pathology Surveillance Center

An Update from Stephen M. Sergay, MB, BCh & Pierluigi Gambetti, MD

April 3, 2008

http://www.aan.com/news/?event=read&article_id=4397&page=72.45.45

In this context, a word is in order about the US testing program. After the discovery of the first (imported) cow in 2003, the magnitude of testing was much increased, reaching a level of >400,000 tests in 2005 (Figure 4). Neither of the 2 more recently indigenously infected older animals with nonspecific clinical features would have been detected without such testing, and neither would have been identified as atypical without confirmatory Western blots. Despite these facts, surveillance has now been decimated to 40,000 annual tests (USDA news release no. 0255.06, July 20, 2006) and invites the accusation that the United States will never know the true status of its involvement with BSE.

In short, a great deal of further work will need to be done before the phenotypic features and prevalence of atypical BSE are understood. More than a single strain may have been present from the beginning of the epidemic, but this possibility has been overlooked by virtue of the absence of widespread Western blot confirmatory testing of positive screening test results; or these new phenotypes may be found, at least in part, to result from infections at an older age by a typical BSE agent, rather than neonatal infections with new "strains" of BSE. Neither alternative has yet been investigated.

http://www.cdc.gov/ncidod/EID/vol12no12/06-0965.htm

Cases of atypical BSE have only been found in countries having implemented large active surveillance programs. As of 1st September 2007, 36 cases (16 H, 20 L) have been described all over the world in cattle: Belgium (1 L) [23], Canada (1 H)15, Denmark (1 L)16, France (8 H, 6 L)17, Germany (1 H, 1 L) [13], Italy (3 L)18, Japan (1 L) [71], Netherlands (1 H, 2 L)19, Poland (1 H, 6 L)20, Sweden (1 H)21, United Kingdom (1 H)22, and USA (2 H)23. Another H-type case has been found in a 19 year old miniature zebu in a zoological park in Switzerland [56]. It is noteworthy that atypical cases have been found in countries that did not experience classical BSE so far, like Sweden, or in which only few cases of classical BSE have been found, like Canada or the USA.

And last but not least, similarities of PrPres between Htype BSE and human prion diseases like CJD or GSS have been put forward [10], as well as between L-type BSE and CJD [17]. These findings raise questions about the origin and inter species transmission of these prion diseases that were discovered through the BSE active surveillance.

full text 18 pages ;

http://www.vetres.org/index.php?option=article&access=standard&Itemid=129&url=/articles/vetres/pdf/2008/04/v07232.pdf

EFSA Scientific Report on the Assessment of the Geographical BSE-Risk (GBR) of the United States of America (USA)

Summary of the Scientific Report

The European Food Safety Authority and its Scientific Expert Working Group on the Assessment of the Geographical Bovine Spongiform Encephalopathy (BSE) Risk (GBR) were asked by the European Commission (EC) to provide an up-to-date scientific report on the GBR in the United States of America, i.e. the likelihood of the presence of one or more cattle being infected with BSE, pre-clinically as well as clinically, in USA. This scientific report addresses the GBR of USA as assessed in 2004 based on data covering the period 1980-2003.

The BSE agent was probably imported into USA and could have reached domestic cattle in the middle of the eighties. These cattle imported in the mid eighties could have been rendered in the late eighties and therefore led to an internal challenge in the early nineties. It is possible that imported meat and bone meal (MBM) into the USA reached domestic cattle and leads to an internal challenge in the early nineties.

A processing risk developed in the late 80s/early 90s when cattle imports from BSE risk countries were slaughtered or died and were processed (partly) into feed, together with some imports of MBM. This risk continued to exist, and grew significantly in the mid 90’s when domestic cattle, infected by imported MBM, reached processing. Given the low stability of the system, the risk increased over the years with continued imports of cattle and MBM from BSE risk countries.

EFSA concludes that the current GBR level of USA is III, i.e. it is likely but not confirmed that domestic cattle are (clinically or pre-clinically) infected with the BSE-agent. As long as there are no significant changes in rendering or feeding, the stability remains extremely/very unstable. Thus, the probability of cattle to be (pre-clinically or clinically) infected with the BSE-agent persistently increases.

http://www.efsa.europa.eu/en/science/tse_assessments/gbr_assessments/573.html

http://www.efsa.europa.eu/etc/medialib/efsa/science/tse_assessments/gbr_assessments/573.Par.0004.File.dat/sr03_biohaz02_usa_report_v2_en1.pdf

Tuesday, June 3, 2008

SCRAPIE USA UPDATE JUNE 2008 NOR-98 REPORTED PA

http://nor-98.blogspot.com/2008/06/scrapie-usa-update-june-2008-nor-98.html

Tuesday, June 3, 2008 SCRAPIE USA UPDATE JUNE 2008 NOR-98 REPORTED PA

http://nor-98.blogspot.com/2008/06/scrapie-usa-update-june-2008-nor-98.html

Thursday, April 03, 2008 A prion disease of cervids: Chronic wasting disease 2008

1: Vet Res. 2008 Apr 3;39(4):41

http://chronic-wasting-disease.blogspot.com/2008/04/prion-disease-of-cervids-chronic.html

Transmissible Mink Encephalopathy TME

http://transmissible-mink-encephalopathy.blogspot.com/

Communicated by: Terry S. Singeltary Sr. <flounder9@verizon.net>

[In submitting these data, Terry S. Singeltary Sr. draws attention to the steady increase in the "type unknown" category, which, according to their definition, comprises cases in which vCJD could be excluded. The total of 26 cases for the current year (2007) is disturbing, possibly symptomatic of the circulation of novel agents. Characterization of these agents should be given a high priority. - Mod.CP]

http://pro-med.blogspot.com/2007/11/proahedr-prion-disease-update-2007-07.html

http://www.promedmail.org/pls/askus/f?p=2400:1001:6833194127530602005::NO::F2400_P1001_BACK_PAGE,F2400_P1001_PUB_MAIL_ID:1010,39963

There is a growing number of human CJD cases, and they were presented last week in San Francisco by Luigi Gambatti(?) from his CJD surveillance collection.

He estimates that it may be up to 14 or 15 persons which display selectively SPRPSC and practically no detected RPRPSC proteins.

http://www.fda.gov/ohrms/dockets/ac/06/transcripts/1006-4240t1.htm

http://www.fda.gov/ohrms/dockets/ac/06/transcripts/2006-4240t1.pdf

2008

The statistical incidence of CJD cases in the United States has been revised to reflect that there is one case per 9000 in adults age 55 and older. Eighty-five percent of the cases are sporadic, meaning there is no known cause at present.

http://www.cjdfoundation.org/fact.html

Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA 77518